The sleep cycle has changed a lot in the past few decades.

But that’s not stopping the sleep scientist from trying to explain it.

In the last few decades, we’ve been inundated with new technologies that offer new opportunities for our bodies to get us through the day.

The new technology of our own making, however, is the sleep cycle.

A few years ago, researchers at the University of Texas and the University at Buffalo found that they could help people get a little more sleep by manipulating the sleep cycles of their brains.

They used a technology that involves using electrodes on the scalp to stimulate neurons in the brain, to help people achieve more REM sleep.

That’s a very short, deep sleep in which we actually fall asleep.

It turns out that if we can use electrodes on our scalp to help our brain, it would be able to stimulate REM sleep, which is a deeper sleep that we need to complete in order to fall asleep again.

And that’s exactly what happened in the study.

The scientists implanted electrodes on a portion of the brain called the suprachiasmatic nucleus (SCN) in the scalp of mice.

They put the electrodes on their scalp and allowed the mice to fall into deep sleep.

They then gave the mice an electric shock to the scalp, which produced an electrical signal.

The scientists then used a brain imaging technique to see how the electrical activity in the SCN was affected by the sleep stimulation.

The electrical activity was then measured using a technique called functional magnetic resonance imaging (fMRI).

The scientists found that when the researchers applied an electric current to the SCNs of the mice, the electrical signal from the electrodes changed in a way that was different from what they were seeing with the brain imaging.

The researchers were surprised by this.

They found that the EEG activity of the SCn neurons in mice was different than that of the normal SCN neurons in other mice.

That meant that the SCNS neurons in that mouse could not only be influenced by the EEG signals of other mice, but also by those of other people.

The fact that this was happening in mice, as opposed to humans, means that the brain is a very flexible and adaptable organ.

That means that it can be used to help with everything from staying awake at night to sleeping through the night.

The team then showed that it could be used for a lot more than just sleep.

For instance, they could potentially change the electrical signals from the SCNN neurons to improve the efficiency of other brain areas, such as the limbic system, which has been shown to be important for the control of emotions.

In other words, the researchers could potentially help us sleep through the entire night, because our brains are so flexible, adaptable and flexible-minded.

And they found that by changing the electrical stimulation, they had the potential to change the whole sleep cycle and therefore the sleep quality of the mouse.

It’s a remarkable finding, because it’s the first time anyone has shown that EEG stimulation can help you to improve your sleep.

And it also shows that it is possible to have an effect on other brain regions in the body, which could potentially be helpful for people in the long run.

But the team’s next goal was to test whether they could also increase REM sleep by stimulating REM neurons in another brain area, the amygdala.

The amygdala is a region in the human brain that plays a key role in emotion regulation.

It also regulates the emotions we experience in the first place, and the amygdala is particularly vulnerable to emotional stressors.

The reason why this region is particularly sensitive to emotional distress is that it’s also involved in a process called the regulation of emotion processing, which means that when we experience a stressor, our amygdala sends a signal to other parts of the body that helps us to regulate our response to that stressor.

But this is a process that happens all the time.

For example, when we are anxious or stressed, our body sends out a signal called cortisol to try to calm us down.

And the hypothalamus, which controls the release of the hormone cortisol, sends a similar signal to the amygdala to try and ease our discomfort.

If the hypothalamic-pituitary-adrenal (HPA) axis is off, our immune system is not functioning well, and our ability to regulate emotions is also impaired.

In order to increase the activity of REM neurons that are part of the HPA axis in the amygdala, the scientists had to stimulate the activity in another part of this circuit called the amygdala-thalamo-cortical axis.

This circuit is responsible for emotional responses, and it’s a key part of our reward processing.

So by stimulating that circuit, they were able to increase REM activity in these areas of the amygdala and make the animals more alert.

But what is the function of REM sleep?

It’s a sleep cycle in which you fall asleep gradually.

So in the sleep stage, we’re sleeping in a deep